This Article  • Full text  • PDF  • Send to a friend  • Save in My Folder  • Save to citation manager  • Permissions  Citing Articles  • Contact me when this article is cited  Related Content  • Related article  • Similar articles in this journal  Social Bookmarking   What's this?

Arch Surg. 2007;142(11):1104.

	Since this article does not have an abstract, we have provided the first 150 words of the full text and any section headings.

Answer: PH Secondary to Unilateral Adrenal Hyperplasia

The patient underwent transperitoneal laparoscopic left adrenalectomy, which was uncomplicated. Final histologic examination of the left adrenal gland demonstrated cortical hyperplasia involving the zona glomerulosa, fasciculata, and reticularis. There was a nodular proliferation of cells that formed nested groups and trabeculae with round ovid nuclei and vesicular to clear cytoplasm. These features were compatible with adrenal hyperplasia. At a follow-up of 12 months postoperatively, the patient was cured of hypokalemia with a serum potassium concentration of 4.2 mEq/L without supplements. Her hypertension was not cured but she had improved control. Her blood pressure was 130/80 mm Hg with 50 mg of atenolol every morning and 30 mg of nifedipine every morning.

Primary hyperaldosteronism is one of the most common causes of secondary hypertension. It was first described in 1955 by Conn¹ as a syndrome characterized by hypertension, hypokalemia, and excessive urinary excretion of aldosterone secondary to an adrenal cortical adenoma. . . . [Full Text of this Article]

AUTHOR INFORMATION



CiteULike    Connotea    Del.icio.us    Digg    Reddit    Technorati    Twitter     What's this?

RELATED ARTICLE

Image of the Month—Quiz Case
Brian K. P. Goh, Yeh-Hong Tan, Jane Tran, Sidney K. H. Yip, and Christopher W. S. Cheng
Arch Surg. 2007;142(11):1103.
EXTRACT | FULL TEXT